Louisa Enright's Blog

Mainely Tipping Points

Posts Tagged ‘statins

Interesting Information: Osteoporosis Cure

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Interesting Information:  May 21, 2013

Osteoporosis Cure

I’m behind in my reading and reporting.

Blame it on the inherited ipad where I am playing “Word” with kin, friends, and at least one former highschool classmate.  It will keep my brain active, right?  And it allows me to stay connected in a whole new way, right?  I hope so, as I love language and words and am learning so many new ones.

I finished the winter 2012 WISE TRADITIONS, the journal of The Weston A. Price Foundation, the other day.  There is always such good information in it, and it’s free on-line to any reader.  (I get a hard copy because I write all over the pages taking side notes, making comments, circling important information, and so forth.)  This issue is on the importance of fat-soluble vitamins–and I will write more on that tomorrow.   Remember that I am reading so I can report back to you and if you want to read more, you can follow in my footsteps and go to the texts I surface for you.

A letter called “Geriatric Rickets” caught my eye, written by Philip Ridley of London, UK.  His mother suffered from osteoporosis–a disease he believes (as I do) that is caused by malnutrition from the diet his (and our) health practitioners have been pushing for the past forty years or so–low-fat, high-sugar, high-carb intake.

First, Ridley’s mother stopped taking the osteoporosis drugs “given for free in the U.K. on the National Health Service.”  Ridley notes that

these drugs operate by inhibiting osteoclasts and stimulating osteoblasts.  The former break down old bone cells and the latter build new bone cells.  The problem with meddling in this process is that strong bones require the renewal of old bone cells with new bone cells.  The drugs therefore increase brittleness and they also do nothing about the malnutrition that causes weak bones in the first place.”

Ridley also notes that “women at the final stages of geriatric rickets are given an infusion of these toxic drugs directly into the marrow.  I have heard from families that this is the most painful treatment.”

Ridley’s mother CURED her osteoporosis by eating “bone broths, sourdough bread [fermented foods], butter, soaking of beans and grains, raw grass-fed Guernsey milk, two Royal Blend high-vitamin butter oil and fermented cod liver oil capsules per day, liver and bacon once a week, and an herbal remedy for strong bones.”  Ridley’s mother “had always had grass-fed meat, wild fish, and fresh vegetables, but lacked the fat-soluble vitamins as a result of following the lowfat diet since it was introduced into Britain in 1983, when skimmed milk first came available.”

Ridley and his mother spent “the last decade since her diagnosis waiting for the horrid, inevitable broken hip or back bone.”  But, Ridley reports that her last bone density test showed that she no longer needed to be followed for osteoporosis.  Her diet had healed her bones.

Ridley also notes that the only nutritional supplement  given for osteoporosis in the UK is calcium tablets.  But, calcium given this way “simply calcifies the soft tissues in combination with the low fat diet they promote.”  When people ask Ridley how to strengthen bones he says “eat bones.”

Ridley dams the way doctors and Big Pharma work together to put women on drugs–and what he says is true in America as well:

Geriatric rickets is becoming a silent, worsening epidemic amongst women because the bone density tests kick in for all at around sixty-five years of age, and, much like the cholesterol levels that lead to statin prescriptions, the triggers for bone density treatment are manipulated to catch the greatest number of customers for the drug companies.”

AND:

Doctors in the NHS also get performance-related pay based on the number of women tested and the number of women who test negative who hare placed on the drugs.  Most women nowadays will, as a result of lowfat diets, suffer low bone density, so a vast number of women are now being put on these toxic drugs, yet they could all be saved anguish if we would only call osteoporosis what it is and treat it accordingly.

That would be “geriatric rickets.”

Ridley also notes that “routine bone density tests most likely also cause cancer because they use radiation.”

I could add that when I came to Maine, I had arthritis in my right hip and terrible back pain.  I know my bones are much stronger now as a result of how I eat.  My gums don’t bleed when I go to the dentist.  And I’ve (knock on wood) had no new cavities–a sure sign of malnutrition.  I refuse to get any more bone density tests.  Or, mammograms, for that matter.  And I’m not going to go through the airport x-ray machines any more either.

I also have well water, which means I am no longer getting any fluoride.  For about two years after we came to Maine, I could hardly sleep at night from the pains in my bones.  I was restless and twisted and turned.  I just plain hurt.  I think it was the fluoride coming out of  my bones–and fluoride has been shown to make bones brittle, not strong.  There are a number of essays on this blog addressing the fluoride–which is one of the biggest scams in our lives today.  There isn’t any science behind adding it to the water and a LOT of science showing how dangerous it is.  Anyway, I don’t have these pains any more, and I can feel such an improvement in the health of my bones.

Here’s the whole letter if you want to read it:  http://www.westonaprice.org/letters/letters-winter-2012.

Mainely Tipping Points 37: Statins: Profitable Toxins

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Mainely Tipping Points 37

STATINS:  PROFITABLE TOXINS

 

Stephanie Seneff is a senior research scientist in the EECS (Electrical Engineering, Computer Science) department at the Massachusetts Institute of Technology (MIT).  Her degrees–a B.S. in biology, and an M.S., E.E., and Ph.D. in EECS—were awarded by MIT.  She researches within the interdisciplinary intersections of medicine, computer science, and electrical engineering, or the highly-respected biomolecular discipline.   

Seneff’s article, “Cholesterol:  The Essential Molecule–and The Adverse Effects and Overuse of Statins” (Well Being Journal, November/December 2011, 13-24), is the most complete, chemical explanation I have read of why statins are not a solution to the prevention of heart attacks. Statins, Seneff explains, create a situation where muscles are destroyed and where, eventually, the whole body is seriously at risk. 

Once again, drug industry researchers and medical doctors only looked at one piece of an illness puzzle—prevention of heart attacks–without understanding the actual causes and without acknowledging the long-term impact of their drug (statins) solution.  (Surely they know the harm statins do and are ignoring this harm because statins are so profitable.)  After exhaustive research, Seneff says the following:  “I will…make the bold claim that nobody qualifies for statin therapy, and that statin drugs can best be described as toxins” (13).  And, “I would in fact best characterize statin therapy as a mechanism to allow you to grow old faster” (22).

In addition, the drug industry and doctors have played a game I think of as “medical math.”  Seneff notes that a meta-study reviewing seven drug trials and 42,848 patients over a three- to five-year period did show a 29 percent decreased risk of a major cardiac event.  But as heart attacks were “rare among this group, what this translates to in absolute terms is that 60 patients would need to be treated for an average of 4.3 years to protect one of them from a single heart attack.  However, essentially all of them would experience increased frailty and mental decline….” (14).       

Seneff’s article describes the chemical components within the body when cholesterol is fully present and when it has been compromised.  Her explanations are clear and fully understandable, but complicated.  If you are taking statins or are contemplating them, I urge you to read Seneff’s article.  Meanwhile, I will do my best to synthesize the high points so that you can understand why it is so dangerous to use statins to reduce cholesterol in your body. 

Furthermore, many, many studies—some of them long-term studies—clearly show that people—and especially women–with high cholesterol counts live longer than those with low cholesterol counts.  This information is readily available, and it is a mystery to me why our doctors continue to ignore it.

 Statins interfere with the synthesis of cholesterol, a nutrient, explains Seneff, that has been demonized by the drug industry and doctors, but which is essential to human health.   “Cholesterol is absolutely essential to the cell membranes of all our cells, where it protects the cell not only from ion leaks but also from oxidation damage to membrane fats” (14).  Reducing cholesterol “places a much bigger burden on the body to synthesize sufficient cholesterol to support the body’s needs, and it deprives us of several essential nutrients” (14).       

Further, Seneff notes, “there are three distinguishing factors that give animals an advantage over plants:  a nervous system, mobility, and cholesterol.”  Cholesterol, which is “absent from plants, is the key molecule that allows animals to have mobility and a nervous system” (14). In a nutshell, when statins reduce cholesterol, they force the body to jerry-rig alternative chemical systems that lead eventually to body-wide damage (20).

One mythology today is that elevated serum levels of LDL (low density lipoprotein) cholesterol is a problem.  But, Seneff explains, “LDL is not a type of cholesterol, but… [is] a container that transports fats, cholesterol, vitamin D, and fat-soluble anti-oxidants to all the tissues of the body.”  Because these nutrients are not water-soluble, they “must be packaged up and transported inside LDL particles in the blood stream.”  Thus, “if you interfere with the production of LDL you will reduce the bioavailability of all these nutrients to your body’s cells” (15).

The LDL package, explains Seneff, is “vulnerable to attack by glucose and other blood sugars, especially fructose.”  If “gummed up” by sugars, “the LDL particles become less efficient in delivering their contents to the cells,” they “stick around longer in the bloodstream,” and the “measured serum LDL level goes up” (15).  But, worse, after the LDL particles have delivered their contents, they “become small dense LDL particles, remnants that would ordinarily be returned to the liver to be broken down and recycled.”  However, “the attached sugars interfere with this process…so the task of breaking them down is assumed instead by macrophages in the artery wall and elsewhere in the body.”  These “small dense LDL particles become trapped in the artery wall so that the macrophages can salvage and recycle their contents, and this is the basic source of atherosclerosis” (15). 

The liver, explains Seneff, produces the LDL particles.  Statin therapy “greatly impacts the liver, resulting in a sharp reduction in the amount of cholesterol it can synthesize.”  Also, the liver breaks down fructose and converts it into fat.  So, when there is a lot of fructose in the system, the liver becomes burdened with the task of converting it to fat and cannot “keep up with the cholesterol supply.”  Both conditions mean that “fats cannot be safely transported”(16).

Additionally, as the liver is burdened with handling the fructose, “it produces low quality LDL particles” (16).  So, harmful chain reactions begin to occur, such as the following:  fructose builds up in the blood stream, which causes more damage; the skeletal muscle cells are severely affected; and the brain, which houses 25 percent of the body’s cholesterol, is impaired.  Diabetes and arthritis are also associated with statin therapy (19, 21).   

When overburdened, the liver shifts the processing of excess fructose to the muscle cells, explains Seneff.  The muscle cells themselves begin to use an alternative fuel source that requires an abundance of fructose and which allows the production of lactate, which is a high-quality fuel for the heart.  This desperate production of lactate is why statin therapy can lead to a “reduction in heart attack risk.” (17).

But, continues Seneff, “the muscle cells get wrecked in the process” (17).  In effect, the muscles “can no longer keep up with essentially running a marathon day in and day out.”  The muscles “start literally falling apart, and the debris ends up in the kidney, where it can lead to the rare disorder rhabdomyolysis, which is often fatal” (20).  The drug industry readily admits to muscle pain and weakness with statin use (17).

The dying muscles also “expose the nerves that innervate them to toxic substances, which then leads to nerve damage such as neuropathy, and ultimately amyloid lateral sclerosis (ALS), also known as Lou Gehrig’s disease, a very rare, debilitating, and ultimately fatal disease that is now on the rise due (I believe) to statin drugs” (20).

Also, as the cells struggle with ion leaks caused by insufficient cholesterol, they begin to replace a potassium/sodium system with a calcium/magnesium-based system.  The result is the “extensive calcification of artery walls, heart valves, and the heart muscle itself.”  Indeed “research has shown that statin therapy leads to increased risk of diastolic heart failure” (20). 

Seneff is very interested in the role of cholesterol sulfate.   Cholesterol sulfate is “very versatile.  It is water soluble, so it can travel freely in the blood stream, and it enters cell membranes ten times as readily as cholesterol, so it can easily resupply cholesterol to cells” (24).

Cholesterol sulfate, explains Seneff, is produced by the skin in large quantities with sun exposure.  Seneff  thinks that “the natural tan that develops upon sun exposure offers far better protection from skin cancer than the chemicals in sunscreens.”  And, Seneff thinks we should eat foods “rich in both cholesterol and sulfur”—“eggs are an optimal food, as they are well supplied with both of these nutrients” (24).     

To avoid heart disease, Seneff suggests cutting back on fructose intake, eating whole foods instead of processed foods, and eating foods which are good sources of lactate (sour cream, yogurt, and milk products in general).  (One can use goat-milk products if cow’s milk is a problem.)  Strenuous physical exercise helps “get rid of any excess fructose and glucose in the blood, with the skeletal muscles converting them to the much coveted lactate” (23) 

Seneff further advises:  “spend significant time outdoors; eat healthy cholesterol-enriched, animal-based foods like eggs, liver, and oysters; eat fermented foods like yogurt and sour cream; eat foods rich in sulfur like onions and garlic.  And, finally say `no-thank-you’ to your doctors when they recommend statin therapy” (24).